Beth E. Snitz, PhD

  • Associate Professor
  • Co-Associate Director, Clinical Core, Alzheimer Disease Research Center

Dr. Snitz is a neuropsychologist with research expertise in risk factors for dementia and cognitive decline in aging. She is involved in clinical, neuroimaging and epidemiologic studies investigating cognitive aging across the normal and pathologic spectrum. Currently, Dr. Snitz leads an R01 grant examining PET neuroimaging biomarkers and longitudinal cognitive decline in a population-based sample of older adults without dementia. 

Education & Training

  • BA, Oberlin College, Biopsychology
  • PhD, University of Minnesota -Twin Cities, Clinical Psychology
  • Intern, Ann Arbor VAMC, Psychology
  • Post Doc Resident, Ruhr University Bochum, Germany, Medicine
  • T-32 Post Doc, Department of Psychiatry, University of Pitt. fMRI

Specialized Areas of Clinical, Research and/or Educational Interests 

  • Research: Early cognitive changes associated with preclinical and prodromal Alzheimer Disease; cognitive correlates of Alzheimer biomarkers; risk and protective factors for mild cognitive impairment and dementia; subjective cognitive decline.

Professional Organization Membership

  • American Psychological Association, Division 40, Society for Clinical Neuropsychology
  • International Neuropsychological Society
  • Editorial Board - Journal of the American Geriatrics Society
  • Scientific Advisory Committee - Society for Clinical Neuropsychology

Honors & Awards

  • NARSAD Young Investigator Award
  • NIH K-23 Career Development Award, National Institute on Aging

Selected Publications

Snitz BE,  Chang Y, Tudorascu DL, Lopez OL, Lopresti, DeKosky ST, Carlson MC, Cohen AD, Kamboh MI, Aizenstein HJ, Klunk WE, and Kuller LH.  Predicting resistance to amyloid-beta deposition and cognitive resilience in the oldest-old. In press, Neurology.

Snitz BE, Tudorascu DL, Campbell E, Yu Z, Lopresti BJ, Laymon CM, Mihas DV, Nadkarni NK, Aizenstein HJ, Klunk WE, Weintraub S, Gershon RC and Cohen AD. Associations between NIH Toolbox Cognition Battery and in vivo brain amyloid- and tau-pathology in non-demented older adults. In press,  Alzheimer’s & Dementia: Diagnosis, Assessment and Disease Monitoring.

Goldberg SM, Lopez OL, Cohen AD, Klunk WE, Aizenstein HA, Mizuno A, and Snitz BE. The roles of study setting, response bias, and personality in subjective memory complaints of cognitively normal older adults. International Psychogeriatrics. 2020 ePuB Mar 19:1-12.

Snitz BE, Wang T, Cloonan YK, Jacobsen E, Chang CC, Hughes TF, Kamboh MI, and Ganguli M. Risk of progression from subjective cognitive decline to mild cognitive impairment: The role of study setting. Alzheimer's & Dementia. 2018 Jun 1;14(6):734-42.

Zhao Y, Tudorascu DL, Lopez OL, Cohen AD, Mathis CA, Aizenstein HJ, Price JC, Kuller LH, Kamboh MI, DeKosky ST, Klunk WE and Snitz BE. Amyloid β deposition and suspected non-Alzheimer pathophysiology and cognitive decline patterns for 12 years in oldest old participants without dementia. JAMA neurology. 2018 Jan 1;75(1):88-96.